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1)  mitogen-activated protein kinase
丝裂原激活蛋白激酶
1.
Abstract: Objective To understand the role of p38 mitogen-activated protein kinase (p38MAPK) in the expression of in-ducible nitric oxide synthase (iNOS) and NO production in human endothelial cells under the stimulation by lipopolysaccha-ride (LPS).
目的研究p38丝裂原激活蛋白激酶(p38 MAPK)在脂多糖(LPS)诱导的人内皮细胞－ECV304诱导型一氧化氮合酶(iNOS)表达和一氧化氮(NO)产生中的作用。
2.
Aim:To explore the expression and abnormal activation of mitogen-activated protein kinase (MAPK) in the development and progression of cerebral gliomas.
目的:研究人脑胶质瘤组织中丝裂原激活蛋白激酶(MAPK)的表达及活化情况。
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2)  MAPK
丝裂原激活蛋白激酶
1.
THE ROLE OF MAPK IN LPS-INDUCED iNOS EXPRESSION IN RAT SCHWANN CELLS;
丝裂原激活蛋白激酶在内毒素脂多糖诱导大鼠施万细胞一氧化氮合酶表达中的作用
2.
BACKGROUND AND AIM:To investigate the effects of thioglycolic acid on the activities of MAPK and MPF protein kinases during progesterone-induced Xenopus oocyte maturation process in vitro.
背景与目的:探讨巯基乙酸(TGA)对孕酮诱导爪蟾卵母细胞体外成熟过程中丝裂原激活蛋白激酶(MAPK)和成熟促进因子(MPF)蛋白激酶活性的影响。
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3)  mitogen activated protein kinases
丝裂原激活蛋白激酶
1.
Proliferation of Toxoplasma gondii tachyzoites and its expression of mitogen activated protein kinases in host cells;
弓形虫速殖子在宿主细胞内增殖与丝裂原激活蛋白激酶表达
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4)  MAPKs
丝裂原激活蛋白激酶
1.
AIM To study the changes of ultramicrostructure in atrial myocytes and the expressions of mitogen-activated protein kinases(MAPKs) after rapid pacing.
目的研究快速起搏后心房肌细胞超微结构和丝裂原激活蛋白激酶(MAPKs)表达的变化。
2.
We also found that ALA inhibited LPS-induced phosphorylation of mitogen-activated protein kinases (MAPKs).
这个作用是通过抑制抑制因子-κBα(I-κBα)的磷酸化和降解以及抑制丝裂原激活蛋白激酶的磷酸化调节的。
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5)  mitogen activated protein kinase
丝裂酶原激活蛋白激酶
1.
Objective To investigate the expression changes of mitogen activated protein kinase (MAPK) in Alzheimer s disease (AD) rat model.
目的探讨丝裂酶原激活蛋白激酶(mitogenaetivatedproteinkinase,MAPK)在阿尔茨海默病(Alzheimer’sdiease,AD)发病中的变化及其可能机制。
2.
Objective To investigate the change of mitogen activated protein kinase(MAPK) in the hippocampus of Alzheimer disease(AD) rat model.
目的探讨丝裂酶原激活蛋白激酶(m itogen activated prote in k inase,MAPK)在阿尔茨海默病(A lzhe im er’s d iease,AD)发病中的变化及其可能机制。
6)  p38 mitogen-activated protein kinase
p38丝裂原激活蛋白激酶
1.
Objective:To identify the role of p38 mitogen-activated protein kinase(p38 MAPK)and its specific inhibitor,SB203580,in airway inflammation and mucus hypersecretion in vivo.
目的探讨小鼠吸入丙烯醛后是否出现气道黏液高分泌及其产生机制,观察p38丝裂原激活蛋白激酶(p38MAPK)信号通路在气道炎症损伤、黏液高分泌中的作用,以及应用p38MAPK特异性抑制剂SB203580干预后对气道炎症、MUC5AC的影响。
2.
Objective: To investigate the effects of pentoxifylline (PTX) on activation of p38 Mitogen-activated protein kinase, and to study the protective mechanism of PTX on endothel cell monolayer permeability.
目的:观察己酮可可碱(PTX)对p38丝裂原激活蛋白激酶(MAPK)激活的影响,探讨PTX保护内皮细胞单层通透性的机制。
3.
Though there was one literature about p38 mitogen-activated protein kinase(MAPK) involed in regulation of LPS-induceed ICAM- 1 protein.
尽管已有文献报道p38丝裂原激活蛋白激酶（MAPK）可能参与LPS诱导内皮细胞ICAM-1蛋白的表达，但仍不清楚p38MAPK作用的位点是在ICAM-1蛋白翻译水平或是在基因转录水平。
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补充资料：丝裂原
分子式：
CAS号：

性质：又称丝裂原。系一种化合物，它能促进淋巴细胞的活性，导致可溶因素(例如淋巴激活素、干扰素等)的生产，功能状态的变化和胚细胞的生长。

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