2) Airway mucus hypersecretion
气道黏液高分泌
1.
Effect of simvastatin on airway mucus hypersecretion in rats and its molecular mechanism;
辛伐他汀对大鼠气道黏液高分泌的影响和机制研究
2.
Airway mucus hypersecretion in rats is inhibited by macrolide antibiotics;
大环内酯类抗生素对大鼠气道黏液高分泌模型的干预作用研究
3.
Methods The rat airway mucus hypersecretion was induced by aerosolacrolein inhalation.
目的探讨丙烯醛诱导大鼠气道黏液高分泌发生的机制;了解PPARγ及其配体对气道黏液高分泌过程的调控作用;探讨PPARγ及其配体参与气道黏液高分泌调控的分子机制。
3) mucus overproduction
黏液分泌
1.
Objective: To evaluate the inhibitory effect of Yichuanping capsule (易喘平胶囊) on the airway mucus overproduction in asthmatic mice and its mechanism.
方法:将32只雄性BALB/c小鼠随机分为正常对照组、哮喘组、地塞米松组和易喘平胶囊组(n=8),制备卵蛋白致敏的哮喘模型,高碘酸希夫(PAS)法检测各组小鼠气道黏膜杯状细胞数量及黏液分泌情况,逆转录聚合酶链反应(RT PCR)检测肺组织黏液蛋白MUC5AC mRNA的水平,检测支气管肺泡灌洗液(BALF)中嗜酸粒细胞数及细胞总数,酶联免疫吸附法(ELISA)检测肺组织匀浆液中白细胞介素4(IL4)和γ干扰素(IFNγ)的含量。
4) Chronic mucus hypersecretion
慢性黏液高分泌症
1.
All people completed diagnostic questionnaires for GERD and chronic mucus hypersecretion(CMH)and lung function test.
所有研究对象完成反流性疾病诊断问卷(RDQ)、慢性黏液高分泌症(CMH)问卷和肺功能检查。
5) airway mucus secretion
气道黏液分泌
1.
Conclusion:The down-regulation of AQP5 expression may be involved in airway mucus secretion of asthma.
结论:AQP5蛋白表达下调可能参与了哮喘发病时气道黏液分泌的调节,IFN-γ可上调AQP5蛋白的表达,在哮喘气道黏液分泌中起治疗作用。
6) Mucus hypersecretion
粘液高分泌
1.
Interventions of enalapril on mucus hypersecretion in rat airway epithelium.;
依拉普利对大鼠气道粘液高分泌的干预作用
2.
Aim Establish a novel mouse model of airway goblet cell hyperplasia and mucus hypersecretion.
目的建立一种新的小鼠气道上皮杯状细胞增生和粘液高分泌模型。
补充资料:人的内分泌系统人体主要内分泌腺
李瑞端绘
[图]
说明:补充资料仅用于学习参考,请勿用于其它任何用途。
参考词条