2) focal cerebral ischemia/reperfusion
局灶性脑缺血再灌注
1.
Regulative effect of exogenous NGF on the expression of IL-1β in rat parietal cortex following focal cerebral ischemia/reperfusion;
NGF对局灶性脑缺血再灌注后大鼠皮质IL-1β表达的调控作用
2.
Objective:To observe the effects of the extract of Ginkgo Biloba(EGB) on the expression of glial fibrillary acidic protein(GFAP) of the astrocyte in focal cerebral ischemia/reperfusion injury rats.
目的:观察银杏叶提取物(extract of Ginkgo biloba,EGB)对大鼠局灶性脑缺血再灌注梗死区胶质纤维酸性蛋白(GFAP)表达的影响。
3.
The study was to investigate the effect of nerve growth factor on the expression of CHOP mRNA and protein in hippocampus of rats with focal cerebral ischemia/reperfusion (I/R).
探讨NGF对局灶性脑缺血再灌注后大鼠海马神经元CHOPmRNA及蛋白表达的影响。
3) focal cerebral ischemia/reperfusion
局灶性脑缺血/再灌注
1.
Modification of suture methods for rat model of focal cerebral ischemia/reperfusion;
SD大鼠线栓法局灶性脑缺血/再灌注模型的改进
2.
Objective The suture methods for the establishment of rat model of focal cerebral ischemia/reperfusion is used widely,but problems such as model unstability,low achievement ratio always exist.
目的本研究的目的旨在对传统线栓法制备大鼠局灶性脑缺血/再灌注模型方法进行改良以提高实验成功率。
4) Focal cerebral ischemia-reperfusion
局灶性脑缺血再灌注
1.
Protective effects of osthole on focal cerebral ischemia-reperfusion injury in rats
蛇床子素对大鼠局灶性脑缺血再灌注损伤的保护作用
2.
Objective To explore the difference in dynamic changes of the extracellular amino acids concentrations in ischemia region and remote region during focal cerebral ischemia-reperfusion.
方法颈内动脉插线法制备大鼠大脑中动脉缺血再灌注模型,采用微透析技术取样,高效毛细管电泳-激光诱发荧光法检测,分别观察局灶性脑缺血再灌注过程中缺血脑区(纹状体)和远隔脑区(海马)细胞外3种氨基酸含量的动态变化。
3.
Methods:Focal cerebral ischemia-reperfusion model was made by improved Longa s methods in male Sprague-Dawley rats( 250~300g), which were randomly se.
第一章 依达拉奉对大鼠局灶性脑缺血再灌注后神经功能及梗塞面积的影响 目的: 利用大鼠大脑中动脉缺血再灌注模型,探讨自由基清除剂依达拉奉(3-methyl-1-phenyl-2-pyrazolin-5-one/MCI-186)对大鼠局灶性脑缺血再灌注的梗塞面积及神经功能评分的影响。
5) local brain ischemia and reperfusion injury
局灶性脑缺血/复灌损伤
1.
Aim:To observe the neuroprotective effect of limb ischemic post-conditioning(LIPC) on local brain ischemia and reperfusion injury in rat,and to investigate whether mitochondrial ATP sensitive potassium channel(mito KATP) and κ-opioid receptor were involved in the neuroprotection.
目的:观察肢体缺血后处理(LIPC)在大鼠局灶性脑缺血/复灌损伤中的神经保护作用及其作用机制。
补充资料：短暂性脑缺血发作
短暂性脑缺血发作
transient ischemic attack,TIA
急性脑血管病之一。指一时性脑缺血引起的一种局限性脑功能丧失，通常在24小时内完全缓解，不遗留重要神经功能缺陷。主要病因是脑动脉粥样硬化，亦可见于各种原因的动脉炎和心脏病。颈内动脉系统的脑缺血发作以病灶对侧的单瘫或偏瘫为常见，尤以上肢和面部为重，可伴有失语及精神症状。椎-基底动脉系统的脑缺血发作常见症状有眩晕、复视、构音障碍、吞咽困难、共济失调、单侧或双侧肢体瘫痪或感觉障碍等，至少两种以上症状共同出现。大脑后动脉供血不足可出现皮质盲，对侧同向偏盲。防治短暂性脑缺血发作，应针对每个人的病因，对发作次数多，考虑为微栓塞所致者，可慎重地选择抗凝治疗。主要病灶在颈部的动脉、颈内动脉颅内段或限于大脑中动脉主干者，可结合病人的具体情况考虑外科治疗。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条