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1)  Ganoderma lucidum polysaccharides
灵孢多糖
1.
The effects of Ganoderma lucidum polysaccharides on the activity of microglia in rat primary glia stimulated with lipopolysaccharides and the expression of TNF-a mRNA and iNOS mRNA;
灵孢多糖对脂多糖诱导的原代中脑小胶质细胞激活及TNF-a mRNA和iNOS mRNA表达的影响
2.
Objective To examine the neuroprotective effects of Ganoderma lucidum polysaccharides(GLPS) on 1-methyl-4-phenylpyridinium ion(MPP+) induced neurotoxicity in PC12 cells.
【目的】观察灵孢多糖(GLPS)对甲基-苯基-吡啶离子(MPP+)诱导损伤的PC12细胞的保护作用。
3.
To investigate the effects of Ganoderma lucidum polysaccharides(GLPS) on the protection of rat primary dopaminergic neurons induced by lipopolysaccharides(LPS).
目的观察灵孢多糖对脂多糖诱导的原代多巴胺能神经元变性的保护作用。
2)  Ganoderma lucidum spores polysaccharides
灵芝孢子多糖
1.
Immunomodulatory effects of Ganoderma lucidum spores polysaccharides on tumor-bearing mice;
灵芝孢子多糖对荷瘤小鼠的免疫调节作用
3)  ganoderma lucidum spore polysaccharide
灵芝孢子粉多糖
4)  Saccharopolyspora sp.nov
糖多孢菌
1.
To explore the antitumor active components in ethyl acetate extract of Mycale plumose sponge-derived actinomycete Saccharopolyspora sp.
羽毛山海绵(My ca le p lum ose)来源的糖多孢菌(S accharop oly sp ora sp。
5)  ganoderma lucidum polysaccharide
灵芝多糖
1.
Study on the effects of different culture media on Ganoderma lucidum polysaccharides and their fermentation optimization;
培养基组成对灵芝多糖形态的影响及优化研究
2.
Selection of Optimal Medium of Ganoderma lucidum Polysaccharide Produced by the Way of Solid Fermention;
固态发酵生产灵芝多糖培养基的优化
6)  Ganoderma polysaccharide
灵芝多糖
1.
Optimization of submerged fermentation conditions for Ganoderma polysaccharide;
灵芝多糖液体发酵条件的优化和pH控制策略的研究
2.
Content Determination of Ganoderma Polysaccharide in Lingzhi Pill;
灵芝滴丸中灵芝多糖的含量测定
3.
Ganoderma polysaccharides antagonize prostaglandin E2-induced suppression of murine splenocyte IFN-γ and TNF-α mRNA expression;
灵芝多糖拮抗前列腺素E2对小鼠脾细胞IFN-γ和TNF-α mRNA表达的抑制作用
补充资料:环孢灵
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性质:又名环孢素,环孢灵,山地明。由真菌Tolypocladium inflatum和Cylindrocarpon incidum中分离得到的中性环多肽混合物的主要成分。针状结晶,熔点148~15l℃,旋光度[α]D20-244°(c=0.6,氯仿)溶于甲醇、乙醇、丙酮、氯仿、乙醚,微溶于水。具有抑制免疫系统功能的作用。其作用机理可能是由于它可以选择性地通过阻滞细胞膜上的同种白细胞抗原(HLA-DR)受体,抑制T辅助细胞和细胞毒T细胞所致。口服吸收慢且不完全,平均生物利用度30%,并有较高的蛋白结合率,体内分布广,分布容积为4~13L/kg,主要经肾排泄。用于肾、肝及心脏移植的排异反应。

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