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1)  plasma N-terminal brain natriuretic peptide
N末端脑利钠肽
1.
Objective To evaluate the effect of percutaneous closure of patent ductus arteriosus(PDA)on left ventricular size and function by measuring plasma N-terminal brain natriuretic peptide(NT-proBNP)level and using two-dimensional echocardiography.
目的应用心脏超声技术及检测血浆N末端脑利钠肽(NT-proBNP)水平,探讨经皮动脉导管未闭(PDA)封堵术对心功能影响。
2)  NT-proBNP
N末端脑利钠肽前体
1.
The dynamic change of NT-proBNP in patients with multiple injury at the early stage after trauma and its evaluation value of injury severity;
多发伤患者早期N末端脑利钠肽前体动态变化及其伤情评估价值
3)  N-terminal pro-brain nitric peptide (Nt-proBNP)
N-末端脑利钠肽前体
4)  amino-terminal fragment of the BNP
N末端脑尿钠肽
5)  NTproBNP
N末端前脑钠肽
1.
Our study aims to observe the serum levels of patients with coronary heart disease(CHD) accompanied with congestive heart failure(CHF)and patients with CHD accompanied without CHF,to investigate relationships between the serum levels of adiponectin and NTproBNP and the role of serum adiponectin in patients with CHD accompanied with CHF.
方法收集连续入院的冠心病合并NYHAII-IV级患者60例和冠心病无心功能不全患者60例的血清和临床资料,选取性别与年龄与病例组相匹配对照组25例,测定空腹血清脂联素水平、N末端前脑钠肽(N-terminal portion of proBNP,NTproBNP)、hsCRP(high sensitivity C-Reactive Protein)及生化指标,比较各组间脂联素水平及与NTproBNP、hsCRP及生化指标的关系。
6)  NT-proBNP
N末端脑钠肽
1.
Comparative study on BNP and NT-proBNP in the elderly with diastolic heart failure;
脑钠肽和N末端脑钠肽前体在老年舒张性心力衰竭患者中的比较
2.
Value of NT-proBNP in differential diagnosis of dyspnea which caused by heart disease or pulmonary disease;
N末端脑钠肽在鉴别心源性和肺源性呼吸困难中的应用研究
3.
Objective:To explore the relationship between plasma concentration of big endothelin-1(Big ET-1),N-teriminal probrain natriuretic peptide(NT-proBNP) and left ventricular aneurysm(LVA) after acute myocardial infarction(AMI).
目的:探讨急性心肌梗死(AMI)患者血浆大内皮素(ET)、N末端脑钠肽(NT-proBNP)水平和左室室壁瘤(LVA)形成之间的关系。
补充资料:利钠肽
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性质:又称心房利钠肽。28肽,SLRRSSCFGGRIDRIGAQSGLGCNSFRY分子中包含一个十七肽二硫键内环。主要分布于右心房,含量达0.2g/kg组织,左心房含量为右心房的1/4左右,肺叶血管壁1nmol/kg;下丘脑、中脑、纹状体等约为5nmol/kg。血容量增大使心脏或血管壁受到较大的牵张刺激时,能导致心房肌细胞释放ANF。乙酰胆碱、肾上腺素和加压素通过膜受体磷脂酰肌醇途径促进ANF释放。ANF释入血液后对动脉壁、肾细胞立即通过cGMP-蛋白激酶G调节血容量、水电解质平衡和血压。在脑中有可能作为神经递质参与水平衡调节。

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