1) cerebral hypoperfusion
脑低灌
1.
Effects of cerebral hypoperfusion on thalamic mast cells in female rats;
轻度脑低灌对雌性大鼠丘脑内肥大细胞的影响
2) Cerebral hypoperfusion
脑低灌注
1.
Objective To explore the effects of cerebral hypoperfusion on the tau hyperphosphorylation and impaired cholinergic nervous system in rat brain.
目的探讨脑低灌注对脑内tau蛋白的过度磷酸化和胆碱能指标变化的影响。
2.
Objective:To explore the effect of chronic cerebral hypoperfusion on the expres- sions ofβ-amyloid (Aβ) and apoptosis related indicators in brain tissues.
目的：探讨慢性脑低灌注对脑组织β淀粉样蛋白(Aβ)和细胞凋亡相关指标表达的影响。
3) chronic cerebral hypoperfusion
慢性脑低灌注
1.
Effects of Weinaokang capsule on intracephalic cholinergic system and capability of scavenging free radicas in chronic cerebral hypoperfusion rats;
维脑康胶囊对慢性脑低灌注大鼠脑内胆碱能系统及清除自由基能力的影响
2.
Objective:To observe the intervention and mechanism of Weinaokang Capsule(WNK)on cognitive impairment rats induced by chronic cerebral hypoperfusion.
目的：观察维脑康胶囊(WNK)对慢性脑低灌注所致大鼠认知障碍的干预作用及机制。
3.
Aim:To observe the expressions of Cx32 and Cx36 in the hippocampus of rats suffering from chronic cerebral hypoperfusion.
目的:观察慢性脑低灌注大鼠海马组织中缝隙连接蛋白(Cx)32和Cx36的变化,探讨其在慢性脑低灌注认知功能损害中的作用。
5) chronic cerebrohypoperfusion
脑慢性低灌注
1.
Considerable researches had shown that brain ischemia, particularly chronic cerebrohypoperfusion prevailing in the elderly strongly associated with AD and resu.
近年来,大量研究表明,脑缺血,尤其是老年人普遍存在的脑慢性低灌注与散发性AD强烈相关,可以导致AD发病过程中的核心物质β-淀粉样蛋白(β-amyloid protein,Aβ)增加,但是具体机制仍不清楚。
6) chronic cerebral hypoperfusion
慢性脑血流低灌注
1.
Behavioral and pathologic changes of chronic cerebral hypoperfusion in mice
小鼠慢性脑血流低灌注状态的行为学检测及病理学观察
2.
Objective To investigate the changes of β-amyloid peptide(Aβ) in rats’ blood and brain under the condition of chronic cerebral hypoperfusion.
目的研究慢性脑血流低灌注对大鼠血和脑内β-淀粉样肽(β-amyloid peptide,Aβ)含量的影响。
3.
Until now, the therapies available of white matter lesions induced by chronic cerebral hypoperfusion include symptomatic treatment and controlling risk factors of cardiovascular and cerebrovascular diseases.
缺血性白质脑病的分子病理机制仍不明确,对于慢性脑血流低灌注导致的脑组织能量代谢障碍和神经活性物质代谢障碍造成以白质损伤为主的病理损害的分子机制仍然缺乏清楚认识。
补充资料：低都儿低
1.低而又低，很低。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条