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1)  peripheral neuritis
末梢神经炎
1.
One hundred and eighteen cases of NDDM Ⅱ complicated with peripheral neuritis were treated with Tangshen Decoction(糖神合剂,TD),30 cases treated with anisodamine injection were set up as the control group.
采用中药糖神合剂治疗 型糖尿病并发末梢神经炎 118例 ,并与西药山莨菪碱治疗 30例单盲对照观察 ,均口服格列苯脲控制血糖。
2)  nerve terminal
神经末梢
1.
It has been well established that MDMA is neurotoxic and can result in long-term degeneration of cerebral 5-hydroxytryptamine (5-HT) nerve terminals in many species.
于末次给药后32周采用原位杂交检测5-HT转运体(serotonin transporter,SERT)mRNA 和内源性焦虑物质苯甲二氮(?)结合性抑制物(diazepam binding inhibitor,DBI)的mRNA表达,免疫组织化学染色检测胶质纤维酸性蛋白(glial fibrillary acidic protein,GFAP)的表达,银染观察神经末梢变化。
2.
The purpose of the present study was to explore whether the skin has the connection with the amniotic fluid and the distribution and development of nitric oxide synthase (NOS) positive nerve terminals in the skin of mouse embryo.
为阐明小鼠胚胎期皮肤的神经末梢是否与羊水有接触关系及胚胎期皮肤一氧化氮合酶(NOS)阳性神经末梢的分布和发育状况,利用逆行轴浆运输的原理将荧光金在体注射到胚胎的羊水中,用荧光显微镜观察神经元被标记情况。
3)  nerve endings
神经末梢
1.
Fast silver-staining of nerve endings of muscle spindle in rat soleus muscle;
大鼠比目鱼肌肌梭内神经末梢的快速银染方法
2.
Objective To investigate changes induced by hindlimb unloading in morphology of nerve endings in soleus muscle spindles.
目的观察后肢去负荷对大鼠比目鱼肌肌梭神经末梢形态的影响。
3.
The technique of Fawosky s Staining was used to detect the morphology of nerve endings of soleus muscle spindle in 7 d, 14 d, 21 d tail suspended and control rats.
采用肌梭Faworsk氏显示法 ,光镜观察模拟失重不同时期比目鱼肌肌梭结构和神经末梢形态的改变。
4)  the nerve ending
末梢神经
1.
Objective Periphery the comparison outside the vein and the neck the vein leaving alone needle puncture,after inputting Austria Sha Libo and 5-fluorine uracils,creates the phlebitis and the nerve ending damage observation to the patient.
目的比较周围静脉和颈外静脉留置针穿刺,输入奥沙利铂及5-氟尿嘧啶后,对患者造成静脉炎及末梢神经损伤的观察。
5)  non-insulin dependent diabetes complicated with peripheral neuritis
糖尿病末梢神经炎
6)  diabetes ending neuritis
糖尿病性末梢神经炎
补充资料:末梢神经炎


末梢神经炎
peripheral neuritiz

又称“多发性神经炎”,为两侧肢体远端对称性多发性的末梢神经变性疾病。常由重金属或化学药物中毒、代谢性疾病、营养障碍、感染、血液循环障碍等多种原因引起。主要表现为肢体远端对称性的运动障碍、感觉障碍以及自主神经功能障碍。可呈急性、亚急性或慢性病程。肌电图、神经传导速度,神经活检和实验室检查有助诊断。针对病因治疗为主,辅以维生素B类、肾上腺皮质激素、理疗、针灸、按摩等。
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