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1)  Prenatal hypoxic adaptation
产前缺氧性适应
2)  hypoxia adaption
缺氧适应
3)  anoxia preconditioning
缺氧预适应
1.
Methods The A/R and anoxia preconditioning(APC)models of primary cultured neonatal rat myocytes were established and the expression vector pEGFP-N2/Pim-3 inserted with wild type target gene Pim-3 was transfected into the cardiomyocytes.
方法采用原代培养新生大鼠的心肌细胞A/R损伤和缺氧预适应(APC)保护模型,将已经构建好的pEGFP-N2/Pim-3质粒导入原代培养的乳鼠心肌细胞中,实验结束后测定Pim-3mRNA及蛋白表达水平(RT-PCR、Western blot法)的改变,同时检测培养液中乳酸脱氢酶(LDH)活性、四唑盐(MTT)比色试验测定细胞存活率、TUNEL法检测细胞凋亡。
4)  Hypoxia preconditioning
缺氧预适应
1.
Upregulation angiogenesis expression by hypoxia preconditioning via activation of protein kinase C;
缺氧预适应通过蛋白激酶C途径上调心肌细胞促血管生成因子的表达
5)  hypoxic preconditioning
缺氧预适应
1.
Changes in glycine content in mouse brain during hypoxic preconditioning;
小鼠脑内甘氨酸含量在缺氧预适应中的变化(英文)
2.
The effect of hypoxic preconditioning on the survival time of hypoxia tolerance in mice;
缺氧预适应对小鼠常压耐缺氧存活时间的影响
3.
To explore the molecular mechanism of hypoxic preconditioning, isolate and analysis the differential genes from the hippocampus of hypoxic preconditioned mice, the technology of mRNA differential display was used and 56 bands of differential genes were recovered.
为了研究缺氧预适应的分子机制 ,分离并分析预缺氧预适应小鼠海马组织的差异表达基因 ,运用mRNA差异显示技术 ,共获得 5 6条差异基因片段 ,对其中 1 4条进行纯化、克隆、测序以及BLAST分析 ,其中 9条为已知基因 ,5条可能为未知基因。
6)  Hypoxic postconditioning
缺氧后适应
补充资料:趋性(见生态适应性)


趋性(见生态适应性)


  趋性见生态适应性、昆虫定向。
  
说明:补充资料仅用于学习参考,请勿用于其它任何用途。
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