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1)  staphylokinase Δ N
葡激酶缺失突变体
2)  taphylokinase variant
葡激酶突变体
1.
OBJECTIVE To investigate the status of a staphylokinase variant (K35R,DGR) loaded microspheres and the main factors that influence the stability of encapsulated DGR.
目的研究微球内葡激酶突变体(K35R,DGR)的存在状态和影响其稳定性的主要因素。
3)  tissue-type plasminogen activator deletion mutant
组织型纤溶酶原激活剂缺失突变体
1.
To compare the efficiency and safety of intravenous thrombolysis by two kinds of tissue-type plasminogen activator deletion mutants in a rabbit model of retinal artery occlusion established by photochemical method.
在兔视网膜动脉阻塞模型上研究两种不同的组织型纤溶酶原激活剂缺失突变体(reteplase,r-PA)的溶栓效果,动态观察视网膜动脉阻塞改变情况、检测血液凝血和纤溶指标以及有无出血并发症等,对比评价两种药物的有效性、安全性。
4)  Staphylokinase mutant gene
葡激酶突变体基因
1.
Staphylokinase mutant gene,deleted 18 amino acids in N end was amplified from staphylokinase gene,and ligated into pUC19 vector,then recombinated with PLY 4, an expression vector,and transformed E.
为制备Mr小的葡激酶以便作口服制剂,用PCR技术克隆N末端缺失18个氨基酸的葡激酶突变体基因。
5)  nitrate reductase-deficient mutant
硝酸还原酶缺失突变体
1.
The NP-1 gene was transformed into the nitrate reductase-deficient mutant nrm-4 of Chlorella ellipsoidea via electroporation, and the transgenic alga expressed the active NP-1 were obtained.
本研究利用椭圆小球藻(Chlorellaellipsoidea)硝酸还原酶(nitratereductase)缺失突变体为受体,构建了包含NPTII基因和硝酸还原酶基因两个筛选标记的兔防御素蛋白表达载体,采用电激法将目的基因转入椭圆小球藻硝酸还原酶缺失突变体nrm-4,获得了正确表达防御素蛋白的转基因藻,从而表明通过硝酸还原酶作为筛选标记基因并结合硝酸还原酶缺失突变体可作为较好的小球藻生物反应器生产模式。
6)  deletion mutant
缺失突变体
1.
Two deletion mutants of K5 were designed on the basis of the structure and disulfide bond distribution of K5(Pro451—Ala541).
根据K5蛋白(Pro451—Ala541)的结构特征和二硫键分布特点,设计K5的两个缺失突变体K5 mut1(Cys461—Cys540,保留K5 kringle环3个完整二硫键但去除N端和C端多余氨基酸)和K5mut2(Cys482—Cys535,打开kringle环,只保留2个二硫键)。
2.
A deletion mutant dsPAα1 (dsPK) was constructed and expressed, which only consisted of the kringle and protease domains.
构建、表达了缺失Finger和EGF区的dsPAα1的缺失突变体 (dsPK) ,研究dsPAα1的结构与功能的关系 。
3.
During early passages,plants contain a mixed population of deletion mutants which are located in the coat protein readthrough (CP RT) gene.
在最初几次重复摩擦接种后 ,小麦病株存在野生型SBWMVRNA2和几个不同大小的外壳蛋白通读 (CP RT)基因的缺失突变体。
补充资料:21号染色体部分缺失综合征


21号染色体部分缺失综合征


  病名。即21q-综合征。
  
说明:补充资料仅用于学习参考,请勿用于其它任何用途。
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