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1)  acute-on-chronic liver failure
慢加急性肝衰竭
1.
Clinical analysis of 101 cases with acute-on-chronic liver failure
慢加急性肝衰竭101例临床分析
2.
There was no report on experimental model for acute-on-chronic liver failure, which is most commonly found in China.
慢加急性肝衰竭是我国最常见的肝衰竭类型,但目前国内外尚无此类肝衰竭动物模型的报道。
3.
Acute-on-chronic liver failure(ACLF) refers to an acute liver funtion decompensation based on chronic liver diseases.
慢加急性肝衰竭是指在慢性肝病基础上出现的急性肝功能失代偿,主要由出血或感染诱发,引起一系列病理生理过程,包括全身血液动力学改变、免疫麻痹与全身炎症反应综合征、肝肾综合征、肝性脑病等。
2)  Actute-on-chronic liver fail-ure
慢加急性(亚急性)肝衰竭
3)  Subacute-on-chronic liver failure
慢加亚急性肝衰竭
4)  chronic liver failure
慢性肝衰竭
1.
Analysis of prognosis patients with chronic liver failure using the criterion of the model end-stage liver disease;
慢性肝衰竭患者终末期肝病模型预后判断的分析
2.
Bleeding from esophageal varices is aleading cause of death in chronic liver failure.
研究慢性肝衰竭上消化道大出血患者内镜止血后血浆D-dimer水平的动态变化规律,根据CTP分级和MELD评分进行分组,观察感染与纤溶亢进间、感染与死亡间以及影响死亡的诸因素间的联系以指导临床治疗并辅助判断预后。
3.
Conclusion Chronic liver failure and decompensated cirrhosis patients have acid-base imbalance and hypoxemia.
目的探讨慢性肝衰竭和失代偿期肝硬化患者血气分析变化的临床意义。
5)  Acute liver failure
急性肝衰竭
1.
Gastrointestinal function and pathologic changes in rats of acute liver failure caused by thioacetamide;
TAA致大鼠急性肝衰竭的胃肠功能及病理改变
2.
Experimental study on the therapeutic effect of hepatocyte growth factor from porcine regenerative liver on acute liver failure rats;
猪再生肝肝细胞生长因子治疗急性肝衰竭大鼠的实验研究
3.
Protective effects of four L-cysteine prodrugs on acute liver failure in mice;
四种半胱氨酸前体药对小鼠急性肝衰竭的保护作用
6)  acute hepatic failure
急性肝衰竭
1.
Establishment of pig models of acute hepatic failure induced by drugs;
药物性急性肝衰竭动物模型的建立
2.
Protective effects of granulocyte colony-stimulating factor on acute hepatic failure induced by D-galactosamine/ lipopolysaccharide in mice;
粒细胞集落刺激因子对小鼠急性肝衰竭的保护作用
3.
Effect of melatonin and modafinil on acute hepatic failure in mice;
褪黑素、莫达非尼对小鼠试验性急性肝衰竭的影响
补充资料:肝外阻塞性胆汁性肝硬化


肝外阻塞性胆汁性肝硬化


又称"继发性胆汁性肝硬化。*胆汁性肝硬化的一个类型,继发于肝外胆管阻塞而引起的肝硬化。主要由总胆管结石、手术后胆管狭管、胰头癌、先天性胆管闭锁或缺如等原因所造成。在梗阻上端的胆道由下而上地逐渐扩大迂曲,胆汁淤积,肝内小胆管扩大而破裂,使肝血管受压,胆汁外渗,肝细胞发生缺血、坏死,纤维组织增生,最后形成肝硬化。临床表现为黄疸、肝脾肿大,皮肤粘膜出血倾向,血胆固醇下降等,晚期表现同*肝硬化。解除肝外阻塞原因,本病有治愈的可能性。
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