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1)  opotosis
继发性神经元损伤
2)  Delayed neuronal damage
迟发性神经元损伤
3)  Neuron damage
神经元损伤
1.
Objective to explore the correlation along nerve growth factor、the repairing of neuron damage、mossy fibers sprouting (MFS)in rat hippocampus and the role of NGF in excitability synaptic reorganization circuit in temporal lobe epilepsy induced by kainic acid.
【方法】成年大鼠侧脑室内分别注入NGF正、反义寡核苷酸(antisense oligonucleotide,AS-ODN)和磷酸盐缓冲液(PBS)并使用红藻氨酸(kainic acid,KA)致痫后分别在48h、72h、1w和4w采用普通病理方法、免疫组化和Timm S染色法研究NGF反义寡核苷酸干预所引起的NGF蛋白表达变化与大鼠癫痫海马病理改变、神经元损伤修复和苔藓纤维发芽的关系。
2.
Methods Neuron damage was induced by removal of Dglucose in neuro culture medium.
目的探讨在葡萄糖缺失诱导的神经元损伤中,外源性超氧化物歧化酶和过氧化氢酶的作用。
4)  neuron injury
神经元损伤
1.
Ischemic neuron injury in human hippocampusand and its correlation to Caspase-3,MAP-2 and Tubulin;
人脑缺血后神经元损伤与死亡相关因素的关系
2.
Protective effects of Sibelium on neuron injury in rats;
西比灵对神经元损伤的保护作用
3.
Study of the mechanism of cultured neuron injury mediated by nitric oxide during hypoxia and oxidative stress;
低氧和氧化应激中NO介导培养神经元损伤机理的实验研究
5)  Neuronal damage
神经元损伤
1.
The protective role of NGF on hippocampal neuronal damage induced by scalding was investigated in rats.
烫伤可引起海马神经元损伤、海马组织NO含量升高;NGF对烫伤后海马神经元的损伤有保护作用,并能降低NO含量。
6)  injured neurons
损伤神经元
补充资料:继发性胆汁性肝硬化


继发性胆汁性肝硬化
secondary biliary cirrhosis

各种原因的肝外胆管阻塞或上行性感染引起的肝硬化。肝增大、色深绿、质中等,表面有细颗粒结节。临床主要有梗阻性黄疸及原发疾病的症状,肝功能损害较轻;晚期可出现门静脉高压。如能去除病因,肝硬化可部分恢复。
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