1) excision repair cross complementation-1
切除修复基因
1.
But due to the development of resistance to DDP and it s noxious side-effect during treatment, the use of DDP is limited in clinic, the development of resistance is related tightly with the increasing level of excision repair cross complementation-1(ERCC-1) in tumor s cell, the key to treat tumor is how to decrease the leve.
但由于耐药的产生和自身的毒副作用,限制了顺铂的临床使用,耐药的产生与肿瘤细胞内切除修复基因(ERCC-1)的水平上升有密切相关,如何降低肿瘤细胞内ERCC-1的水平成了治疗肿瘤的一个关键。
3) base excision repair gene XRCC1
碱基切除修复基因XRCC1
4) nucleotide excision repair gene
核苷酸切除修复基因
1.
Objective] To assess the possible association between the expression levels of nucleotide excision repair genes with lung cancer susceptibility.
[目的 ]探讨核苷酸切除修复基因表达水平与肺癌易感性关系。
5) Base excision repair
碱基切除修复
1.
Expression of DNA polymerase β induced by all-trans retinoic acid and dimethyl sulfoxide and change of the base excision repair in human esophageal cancer cells;
诱导分化的人食管癌细胞DNA聚合酶β和碱基切除修复功能的改变
2.
BACKGROUND & OBJECTIVE: Base excision repair (BER) genes play important roles in maintaining genomic stability and their abnormal expression are associated with several cancers.
背景与目的:碱基切除修复基因对于维护基因组稳定性具有重要的作用,其表达异常与多种肿瘤相关。
3.
Most members of the XRCC genes family participated in several DNA repair pathways, including base excision repair, homologous recombination repair and non-homologous end joining.
目前已鉴定的这一基因家族的多数成员均参与几种重要的DNA修复途径,包括碱基切除修复、同源重组修复和非同源末端重接。
6) excision repair
切除修复
1.
BR501,an extremely radioresistant bacterium may contain two nucleotide excision repair pathways: the UV damage endonuclease β(UvsE)-dependent excision repair pathway and the UvrABC-dependent pathway.
BR501的核苷酸切除修复系统,可能包含两条途径:依赖于UV损伤内切酶β(UvsE)的修复途径和依赖于UV损伤内切酶α(UvrABC)的修复途径,其关键酶分别为UvsE(dr1819编码)和UvrABC(A亚基dr1771编码)。
补充资料：巴-斯氏扁桃体挤切除器