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1)  nervous functional deficiency
神经功能缺损(NDS)
2)  neurological deficit
神经功能缺损
1.
ObjectiveTo investigate the characteristics of the ambulatory blood pressure of acute cerebral infarction patients in the first 24 hours from onset and to study the relationship between their neurological deficit and characteristics of blood pressure.
使用美国国立卫生研究院卒中评分(NIHSS)评价患者发病第1日、第5日神经功能缺损情况。
2.
Results:The patient experienced recurrent headache,multifocal neurological deficits and cognitive decline with a relapsing and progressive course.
结果:本例患者有反复发作的头痛、多灶性神经功能缺损、认知功能下降等表现以及复发和渐进病程,临床和辅助检查排除了系统性血管病和其他中枢神经系统疾病,头颅MRI显示颅内多灶性和弥漫性病变,MRA和DSA显示右侧大脑中动脉狭窄。
3.
Meso-lifetime and neurological deficit scores at first stroke and recurrence in two groups were observed and analyzed.
方法:首次发生脑卒中患者184例,根据有无并发代谢综合症而分为单纯组102例和并发组82例,均给予相同的治疗和健康教育,观察2组中位生存时间和首次与再发脑卒中时的神经功能缺损评分,并进行统计学分析。
3)  Neurologic impairment
神经功能缺损
1.
Objective: to investigate the effect of stopping endogenous wind and remove obstruction from collaterals capsule on neurologic impairment and viability of ischemic apoplexy patients.
目的:观察熄风通络胶囊对缺血性中风患者神经功能缺损及生活能力的临床疗效。
2.
Results The average decrease of the scores of neurologic impairment for patients who were treated with the exogenous nerve growth factor at acute stage were significantly higher than that for whom treated at convalescence st.
结果急性期使用外源性神经生长因子的脑梗死患者,其神经功能缺损平均减少分数明显高于恢复期使用的患者(P<0。
3.
aspartate aminotransferase (AST), lactate dehydrogenase (LDH), α-hydroxybutyrate dehydrogenase (α-HBDH), creatine kinase (CK) and its MB isoenzyme (CKMB) level of the 38 patients with cerebral infarction in 7days and at the 14th day after being attacked, and to assess their neurologic impairment at the same time by the Scandinanvi.
方法 :于发病 7d内和第 14天检测 38例脑梗死患者血清心肌酶谱 ,即天门冬氨酸氨基转移酶 (AST)、乳酸脱氢酶 (L DH)、α-羟丁酸脱氢酶 (α- HBDH)、肌酸激酶 (CK)和肌酸激酶心型(CKMB) ,同时进行神经功能缺损程度评分 (SSS) ,并与 2 9例对照组对比分析。
4)  Neurofunction deficit
神经功能缺损
1.
The indexes of hemorrkeology,the neurofunction deficit scoring and daily life capacity of patients were assessed before and after treatm.
方法将60例脑梗死患者随机分为两组,均予低分子肝素、丹参注射液及对症治疗,治疗组加用生脉注射液;治疗前后分别测定血液流变学指标,进行神经功能缺损评分及日常生活能力评价。
2.
The clinical effect was observed, the hemorrheologic and hemodynamic parameters were determined, and the neurofunction deficit scoring and daily life capacity of patients were assessed before and after treatment.
方法:82例老年脑梗死患者随机分为治疗组(用参附液治疗)和对照组(用维脑路通治疗),治疗前后分别测定血液流变学和血流动力学参数,进行神经功能缺损评分及日常生活能力评价,并观察其临床疗效。
5)  neurological impairment
神经功能缺损
1.
Objective:To investigate the efficacy and security of autologous transplantation of bone-marrow mononuclear cells for the treatment of patients with neurological impairment.
5例患者神经功能缺损症状较移植前均有很大程度的改善,其中2例神志不清患者神志恢复,可按吩咐动作;4例患者肌力提高1~3个级别;失语症状均得到一定改善。
2.
Results\ buflomedil HCl could reduce the neurological impairment score, platelet aggreg.
结果国产丁咯地尔可显著降低脑梗死患者神经功能缺损评分、血小板聚集率和血液粘稠度 (P<0 。
3.
Objective To investigate the relationship between the plasma homocysteine level and the ischemic sroke among youths,and observe the relationship between change of plasma homocysteine and neurological impairment in patients with ischemic sroke among youths.
目的研究血浆同型半胱氨酸(pHcy)水平与青年缺血性脑卒中的关系;探讨pHcy水平与神经功能缺损程度的相关性。
6)  neurologic deficit
神经功能缺损
1.
Multivariant gradual regressive analysis of influencing factors on neurologic deficit after acute cerebral infarction
急性脑梗死后神经功能缺损影响因素的多元逐步回归分析
2.
Patients neurologic deficit and ischemic events were observed at 1 month and 3 months in follow-up.
方法对急性缺血性卒中的患者96例,在发病后4、7、10 d进行降压治疗,随访1、3个月,观察患者的神经功能缺损、缺血性事件的发生率。
3.
The neurologic deficits,the levels of LPO,SOD and ET-1 in plasma were observed.
方法将35例脑出血患者随机分为两组,观察生大黄对脑出血患者的神经功能缺损程度评分、LPO、SOD及ET-1水平的影响。
补充资料:神经性膀胱功能障碍
神经性膀胱功能障碍
neurogenicbladder dysfunction

   控制排尿的中枢神经(脑或脊髓)或周围神经受到损害后引起的排尿功能障碍。简称神经原性膀胱。几乎每种神经病变都能影响膀胱的功能,但同一类的神经病变可以在不同病人发生完全不同的膀胱功能改变,原因不清楚。常见的神经原性膀胱病因是:脊髓损伤、多发性硬化、脑血管病变、帕金森氏病、糖尿病、脊膜膨出以及手术引起的神经损伤等。神经原性膀胱一般分为两类:①逼尿肌反射抗进,这类膀胱的逼尿肌对刺激的反应有反射抗进现象。②逼尿肌无反射,这类膀胱的逼尿肌对刺激无反射现象。病变在排尿中心以上引起痉挛性膀胱,表现为容量小、不自由收缩、膀胱内压升高、逼尿肌增厚等病状。骶部脊髓2~4是排尿中心的损伤、糖尿病神经病变等引起无张力膀胱,表现为容量大、内压低、无自主性收缩、外括约肌张力 低下等。
   神经原性膀胱常有排尿困难、排尿不易控制,并有神经损害的各种病状。尿流动力学检查可明确神经原性膀胱的类型。尿检查可明确是否合并感染。肾功能测定可明确有无尿毒症。泌尿系造影可了解有无肾积水及输尿管回流,膀胱形态似宝塔形,伴假性憩室。
   治疗可采用各种非手术或手术方法保护肾脏功能;也可用膀胱导尿、间歇导尿、Ditropan治疗、电刺激排尿等方法。如已有严重肾功能损害,需行尿流改道手术。尿失禁时可采用近年设计的各种应用电极控制排尿的器械。
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