1) Mesangial proliferative glomerulonephritis (or Anti-thymocyte serum induced nephritis,ATSN)
系膜增生性肾炎(或抗胸腺细胞血清性肾炎)
2) MsPGN
系膜增生性肾炎
1.
Comparative study of interference of No1 therapy of chronic nephritis on microstructure of immune molecule in mice with MsPGN;
慢肾1号方对小鼠系膜增生性肾炎免疫分子等超微结构干预的对照性研究
2.
Effect of MACC5 isolated from traditional Chinese medicine on the rats with MsPGN induced by anti-Thy1.1;
中草药提取物MACC5对大鼠抗Thy1.1系膜增生性肾炎模型的疗效观察
3.
Objective To study the changes of AQP-2 and ADH of rats with damp-heat type of MsPGN and the mechanism of retention of damp-heat in the interior.
目的研究系膜增生性肾炎(MsPGN)湿热型大鼠水通道蛋白-2(AQP-2)的表达及抗利尿激素(ADH)的变化及湿热蕴郁的机制,并探讨慢肾蛋白停对其的影响及作用机理。
3) Mesangial proliferative nephritis
系膜增生性肾炎
1.
Empirical Study on Molecular Mechanism of Mesangial Proliferative Nephritis Treated by Manshen NO.1 Prescription;
慢肾1号方治疗系膜增生性肾炎分子机理的实验研究
4) Mesangial proliferative glomerulonephritis
系膜增生性肾炎
1.
Analysis of mesangial proliferative glomerulonephritis treated with method of supplementing qi and yin and removing heat and activating blood circulation;
益气养阴清热化瘀法治疗系膜增生性肾炎的探讨
2.
Expression of monocyte chemoattractant protein-1 and infiltration of monocyte/macrophage in human mesangial proliferative glomerulonephritis;
系膜增生性肾炎中单核细胞趋化蛋白1的表达和单核巨噬细胞的浸润
3.
Therapeutic effects of rosiglitazone on mesangial proliferative glomerulonephritis in rats
罗格列酮对大鼠系膜增生性肾炎的治疗作用
5) mesangioproliferative glomerulonephritis
系膜增生性肾小球肾炎
1.
Protective effects of rosmarinic acid on experimental mesangioproliferative glomerulonephritis by reducing renal TGF-β_1 expression;
迷迭香酸对大鼠系膜增生性肾小球肾炎肾内TGF-β_1表达的影响
2.
Objective To investigate the changes and the clinical significant of serum levels of TNF-α and IL-6 level in patients with mesangioproliferative glomerulonephritis.
目的观察IL-6、TNF-α在系膜增生性肾小球肾炎中的作用。
6) MsPGN
系膜增生性肾小球肾炎
1.
Experimental Research on Treating MsPGN with Shenningshuiwan;
肾宁水丸对大鼠系膜增生性肾小球肾炎的实验研究
2.
Objective The expression of IL-13 in plasma and renal tissue was studied to eluciate the molecular pathogenesis of Mesangial proliferative glomerulonephritis (MsPGN).
目的 探讨系膜增生性肾小球肾炎 (MsPGN)患者血浆及肾组织白细胞介素 13(IL -13)的表达 ,进一步了解MsPGN的分子病理机制 ,为临床治疗探索新的途径。
3.
Objective: To study the expressions and quantitative change of collagen components, such as collagen type I and collegen type IV , in glomerular areas of mesangial Proliferative Nephritis (MsPGN) in adults, to identify the correlations between the quantitative changes of collagen components and the degrees of pathological changes.
目的：观察成人系膜增生性肾小球肾炎(MsPGN)肾小球区Ⅰ型胶原和Ⅳ胶原的含量变化规律，以明确胶原成分含量与系膜增生程度及肾小球之间的关系，探讨免疫组化测定胶原含量对MsPGN病理分度和判定肾小球硬化的作用。
补充资料：缺钾性肾炎
人体长期缺钾引起的肾脏病变。钾是细胞内主要阳离子，和许多细胞功能有密切关系；若病人进食钾盐过少或自胃肠道失钾（如各种原因引起的频繁呕吐及腹泻等）过多,或从肾脏排泄钾盐(如长期使用利钾的利尿药物、大剂量肾上腺皮质激素和肾小管有病变)过多,即可引起缺钾，使许多脏器受其影响。低血钾症时病人感肌肉软弱无力，行动不便，甚至四肢瘫痪，重者呼吸困难、心律紊乱，有早搏，心电图上有缺钾改变。肾脏在缺钾时肾小管细胞分泌钾离子减少，故钠离子同氢离子交换增加，尿液呈中性或偏酸性而体液则呈现碱中毒。肾小管细胞有形态学改变，出现空泡变性，以近曲小管为主。长期缺钾可发生间质纤维化。肾脏功能也有改变，病人常有尿量增加及夜尿，尿比重降低等浓缩功能减退表现，重者像尿崩症。尿常规检查可正常或有少量蛋白质、红细胞、白细胞及管型，长期缺钾易诱发尿路感染，此时尿中白细胞明显增加。治疗要去除诱发缺钾的原因，同时补充钾盐可口服10％氯化钾液每日3～6克，缺钾纠正后肾脏浓缩功能逐渐恢复，但较缓慢，常需数月始接近正常。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条