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1)  infectious cirrhosis
感染性肝硬化
2)  Cirrhosis after hepatitis B virus infection
慢性乙型肝炎病毒感染后肝硬化
1.
cirrhosis (Full name: Cirrhosis after hepatitis B virus infection ) is the sixth canse of ten causes of diseasing death in china .
HBsAg携带者和慢性乙型肝炎病毒感染后肝硬化早期,症状均缺乏特异性,治疗上又无特效药物,因此早期确诊及评估其严重程度,在临床上具有现实意义。
3)  Cirrhotic cardiomyopathy
肝硬化性心肌病
1.
Cirrhotic cardiomyopathy is one of the complications of liver cirrhosis which can be characterized by diastolic dysfunction,high cardiac output and decreased systemic vascular resistance.
肝硬化性心肌病是肝硬化的并发症之一,主要表现为心脏舒张功能减低、心排血量增加、全身血管阻力降低。
2.
Cirrhotic cardiomyopathy is easily overlooked clinically often owing to the lack of clinical manifestations.
肝硬化可并发心功能不全,目前认为是肝硬化特有的一种并发症,即肝硬化性心肌病。
3.
Cirrhotic cardiomyopathy is the term used to describe a constellation of features indicative of abnormal heart structure and function in patients with cirrhosis.
肝硬化患者出现心脏结构和功能异常,被称为肝硬化性心肌病。
4)  Biliary cirrhosis
胆汁性肝硬化
1.
Autoantibodies and Immunologic Function in Primary Biliary Cirrhosis: 20 Cases Analysis;
20例原发性胆汁性肝硬化自身抗体和免疫功能分析
2.
Operation treatment of hepatolthiasis with biliary cirrhosis;
肝胆管结石并胆汁性肝硬化的手术处理
3.
Study on variation of lipidemia and apolipoprotein in posthepatitic cirrhosis and biliary cirrhosis;
病毒性肝炎肝硬化与胆汁性肝硬化的血脂及载脂蛋白分析比较(英文)
5)  alcoholic cirrhosis
酒精性肝硬化
1.
Comparison on hematoblastic relevant parameter from patients with liver cirrhosis after heptitis B and patients with alcoholic cirrhosis;
乙型肝炎肝硬化与酒精性肝硬化患者血小板相关参数变化的比较
2.
The effect of reduced glutathione on alcoholic cirrhosis and erythrocyte deformability;
还原型谷胱甘肽对酒精性肝硬化患者红细胞变形性的影响及疗效观察
3.
The serum leptin in alcoholic cirrhosis and non-alcoholic fatty hepatic cirrhosis patients was obviously higher than that in hepatitis B cirrhosis and hepatitis C cirrhosis patients(P<0.
05);酒精性肝硬化组与非酒精性脂肪性肝硬化组相比较,瘦素水平无明显差异(P>0。
6)  Occult patients with cirrhosis
隐匿性肝硬化
补充资料:慢性活动性乙型肝炎


慢性活动性乙型肝炎
chronic active hepaititis B

  病程超过半年,各项症状(消化道症状如厌食、恶心、呕吐、腹胀、腹泻等;神经症状如乏力、萎靡、头晕、失眠等及肝区痛)明显,肝肿大,质地中等以上,可伴有蜘蛛痣、肝掌、毛细血管扩张或肝病面容,进行性脾肿大,肝功能持续异常,或伴有肝外器官损害,或免疫球蛋白、自身抗体持续升高等特征。病理改变以门脉区周围最为明显,门脉区有大量淋巴细胞、浆细胞及巨噬细胞浸润因而扩大。
  
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