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1)  atrophy fiber
萎缩肌纤维
1.
Numerous studies found that ALS is concerned with neurons apoptosis, the main purpose of our study is to approach the relationship between atrophy fibers in ALS and apoptosis.
运动神经元病变导致骨骼肌失神经支配,出现特征性病理变化----小角化肌纤维、肌纤维肥大、分裂肌纤维、萎缩肌纤维成小群分部、核聚集、靶状纤维、RRF(Ragged Red Fiber)、肌纤维类型群化或肌纤维优势。
2)  fibroatrophy
纤维萎缩
3)  fibroatrophy
[医]萎缩纤维化
4)  muscle atrophy
肌萎缩
1.
Preventive and therapeutic effects of proteasome inhibitor MG-132 on dener-vated muscle atrophy of rats;
蛋白酶体抑制剂MG-132对大鼠失神经肌萎缩的防治作用
2.
Rabbit masseteric muscle atrophy resulting from triamcinolone for injection;
曲安奈德肌内注射诱导兔咬肌萎缩的实验研究
3.
Objective To investigate the intervention of ciliary enurotrophic factors(CNTF)on weightlessness-induced muscle atrophy and its myofiber phenotype transition.
目的探讨CNTF对失重性肌萎缩及其纤维表型转换的干预作用。
5)  Muscle atrophy
肌肉萎缩
1.
Purpose To study limb immobilization influencing on denervated muscle atrophy.
结论 肢体制动加速了失神经支配肌肉萎缩的发展。
2.
This paper reviews two main signaling pathways in order to introduce the key role of Akt in it,one is Akt1/FOXOs/MAFbx/MuRF1 signaling path- way which associated with muscle atrophy;the other is PI3K/Akt/mTOR/S6K1 signaling pathway which associated with muscle hypertrophy.
综述与骨骼肌质量变化相关的两个主要信号通路:一方面是与肌肉萎缩相关的Akt1/FOXOs/MAFbx/MuRF1信号通路;另一方面为与肌肉肥大相关的PI3K/Akt/mTOR/S6K1信号通路,从两方面介绍Akt在骨骼肌质量变化中所处的核心地位。
3.
Methods: In order to establish the cell model of using GCs to cause the muscle atrophy,we examined the total protein of the C2C12 muscle tube which was fed with Dexamethasone(DEX,100 nMs) for 1 to 4 days using BCA method.
目的:建立高剂量或长期服用糖皮质激素会引起肌肉萎缩的细胞模型。
6)  Muscular atrophy
肌萎缩
1.
SMN gene diagnosis of spinal muscular atrophy in children;
SMN基因诊断小儿脊髓性肌萎缩症
2.
MRI features of neurogenic muscular atrophy;
神经源性肌萎缩的MRI表现
3.
Molecular diagnosis of puerile spinal muscular atrophy (with report of 10 cases);
儿童进行性脊肌萎缩症的分子诊断(附10例报告)
补充资料:碳(石墨)纤维增强体(见碳纤维、石墨纤维)


碳(石墨)纤维增强体(见碳纤维、石墨纤维)
earbon(graPhite)fibrereinforeements

,、、里z耽维增强体earbon(graphi现inforcements用于复合材料中承受负荷,田的磷纤始知五史纤俯亡e)fibre起增强作
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