1) 3-α-hydroxysteroid dehydrogenase
3-α羟基类固醇脱氢酶
2) 11β-hydroxysteroid dehydrogenase
11β-羟基类固醇脱氢酶
1.
The expression of 11β-hydroxysteroid dehydrogenase in diabetes mellitus vasculopathy treated with different concentration of glucose;
不同浓度葡萄糖对血管内皮细胞11β-羟基类固醇脱氢酶基因表达的影响
2.
Correlation between decrease of 11β-hydroxysteroid dehydrogenase activity and hypokalemia induced by furosemide in rats;
速尿对大鼠肾11β-羟基类固醇脱氢酶活性的抑制作用与其低血钾的相关性(英文)
3.
Expressions of 11β-hydroxysteroid dehydrogenase Types 1 and 2 in colorectal cancer cells and effects of glucocorticoid and glycyrrhetinic acid on this cell line
11β-羟基类固醇脱氢酶在人肠癌细胞株中的表达及甘草次酸的作用
3) 3β-hydroxysteroid dehydrogenase
3β-羟基类固醇脱氢酶
1.
The testis and body weights,testicular histopathology and the activity of 3β-hydroxysteroid dehydrogenase(3β-HSD) of the neonatal mice were investigated.
d)]灌胃作用于怀孕12 d到产后3 d(GD12~PND3)的KM母鼠,观察DEHP对新生雄性仔鼠体重、睾丸重量、Leyd ig细胞形态结构和3β-羟基类固醇脱氢酶(3-βHSD)活性、酶反应面积的影响。
2.
The level of corticosterone of plasma were determined by radioimmunoassay,the 3β-hydroxysteroid dehydrogenase(3β-HSD) .
用放射免疫分析法测定血浆皮质酮;用酶组织化学测定肾上腺琥珀酸脱氢酶(SDH)、3β-羟基类固醇脱氢酶(3β-HSD)的活性。
3.
Objective To investigate the constitution of cells population, changes in 3β-hydroxysteroid dehydrogenase (3β-HSD) expression and androgen productivity in rat Leydig cells cultured by differential adhesion during 4 days primary culture.
目的探讨通过差速贴壁法获得的7天龄、3周龄大鼠睾丸间质内细胞的细胞群体构成、体外培养不同时间3β-羟基类固醇脱氢酶(3β-HSD)的表达水平变化,以及细胞睾酮分泌功能变化。
4) 11β-hydroxysteroid dehydrogenase 2 gene
11β-羟类固醇脱氢酶基因
5) 3β-HSD
3β-羟基类固醇脱氢酶
1.
Objective To explore the effect of Di(2-ethylhexyl)phthalate(DEHP) on testosterone secretion and activity of 3β-hydroxysteroid dehydrogenase(3β-HSD) in Leydig cells cultured from the embryonic mouse testis.
目的探讨邻苯二甲酸二(2-乙基)己酯[D i(2-ethylhexyl)phthalate,DEHP]对体外培养的小鼠胚胎睾丸间质细胞(Leyd ig细胞)3β-羟基类固醇脱氢酶(3-βHSD)活性及睾酮合成的影响。
2.
Methods:The primary rat Leydig cells were cultured 24 h in vitro,and then treated with GnRHa(10-7 mol/L) at varied time courses(6,12,24,36,48 h);the expression of 3β-HSD protein was analyzed by Western blot.
方法:原代培养大鼠睾丸间质细胞,GnRHa(10-7 mol/L)分别处理间质细胞6、12、24、36、48 h后,Western blot方法分析3β-羟基类固醇脱氢酶(3β-HSD)蛋白的变化;MEK1/2抑制剂PD98059(50μmol/L)和GnRHa共作用于间质细胞后,分析3β-HSD蛋白表达和睾酮水平的变化。
6) 11-β hydroxysteroid dehydrogenase type 2
11β羟基类固醇脱氢酶2
补充资料：3-β羟类固醇脱氢酶缺乏症
3-β羟类固醇脱氢酶缺乏症
病名。亦称Δ5-3β-羟类固醇脱氢酶缺陷症。为先天性肾上 腺皮质增生的一种类型，较少见。此酶缺乏，醛固酮、氢化可 的松和雄酮的合成均受阻，表现为肾上腺皮质功能不全。在睾丸亦由于睾酮分泌障碍表现为 功能低下。呈男性假两性畸形，患儿几乎都有隐睾、阴囊分裂和不同程度的尿道下裂。受累 儿童多在出生后不久就出现明显的失盐症状。可采用激素疗法及手术治疗。
说明：补充资料仅用于学习参考，请勿用于其它任何用途。
参考词条